1.
Invitation & Diagnostics
You recommend diagnostic screening to your patients (e.g., before surgery, for chronic diseases, as an additional service).
Diagnostics for Better Recovery
Diagnostics as Prevention – together with you for enhanced regeneration capabilities
Bring preventive diagnostics into your practice. ORY Praevent supports recovery and treatment outcomes through data-based analysis and app feedback.
Diagnostics for regenerative medicine and prevention.
Many medical procedures, therapies and chronic illnesses demand more of the body than its current capacity to perform and regulate. A shortfall in physical, metabolic or physiological capacity raises the risk of complications and lengthens recovery.
A targeted diagnostic screen carried out beforehand makes it possible to identify critical weak points, intervene specifically and improve the capacity to recover. Your practice can thereby take on a valuable preventive role – supporting the medical treatment and increasing patient safety.“
Your Added Value as a Partner
Based on the data, we support your patients with evidence-based recommendations (nutrition, exercise, micronutrients, stress management), matched to their starting point
You receive sound diagnostic data on your patients (e.g. blood, laboratory and vital parameters, metabolic, inflammatory or recovery markers)
You strengthen your medical quality and your distinctiveness by adding a further preventive service
How it Works
2.
Analysis & Feedback
The samples/readings are evaluated → the data is made accessible via app (for the patient and you).
3.
Support & Optimization
We provide concrete recommendations for action, your practice can adapt individually based on the data, and further diagnostics can be performed over time.
"Who is this useful for?"
- Dentists, ahead of major procedures (implants, jaw reconstruction and the like)
- Surgeons (orthopaedics, visceral surgery, cardiovascular)
- Doctors with chronically ill patients (e.g. metabolic, autoimmune, chronic inflammatory conditions)
- Therapists who support patients over the long term (e.g. physiotherapy, naturopathy, complementary medicine)
*Evidence-based research shows: nutrient deficiencies slow healing and hinder the body's recovery.
Deficiencies slow healing and tissue regeneration. Micronutrients are essential for building cells, producing energy and mounting an immune response. Studies show that targeted supplementation and dietary correction support recovery. Preventive diagnostics make it possible to spot this and act in good time. - short, scientific, with a compact summary of the studies
A lack of B vitamins
(particularly B1, B6, B12) impairs nerve regeneration, since these vitamins are needed for myelination, energy metabolism and antioxidant defence. Without them, lasting nerve degeneration, pain and peripheral neuropathy follow.
Lack of vitamin D
Vitamin D deficiency is associated with muscle weakness, reduced muscle regeneration and a higher risk of falls; supplementation improves muscle regeneration after injuries and operations. is associated with muscle weakness, reduced muscle regeneration and a higher risk of falls; supplementation improves muscle regeneration after injuries and operations.
Lack of vitamin E
Vitamin E deficiency leads to impaired nerve regeneration and a reduction in the muscle cell types needed for repair.
Lack of vitamin A
Vitamin A deficiency alters the composition of the extracellular matrix and impairs cell proliferation and differentiation, which disrupts tissue repair.
Amino acid deficiency
Amino acid deficiency – particularly of essential and conditionally essential amino acids such as arginine and glutamine – limits protein synthesis, inhibits cell proliferation and impairs immune function. The result is delayed or incomplete regeneration of muscle, nerve and organ tissue.
Cardiovascular complications:
Raised homocysteine levels with B12 or folate deficiency are associated with a higher risk of thromboembolic events.
Thiamine deficiency can lead to Wernicke's encephalopathy and beriberi, vitamin E deficiency to spinocerebellar ataxia, vitamin A deficiency to visual disturbance and vitamin D deficiency to osteomalacia.
Neurological disorders:
A vitamin B12 deficiency can lead to sensory ataxia, peripheral neuropathy, myelopathy (subacute combined degeneration of the spinal cord), cognitive impairment and psychiatric symptoms. These neurological manifestations can precede the haematological symptoms and are potentially irreversible if not treated in time.
Haematological complications: vitamin B12 and folate deficiency cause megaloblastic anaemia with increased susceptibility to infection, fatigue, breathlessness and, in severe cases, pancytopenia.
CNS disorders, weakened immunity and impaired wound healing:
CNS disorders: a deficiency of certain amino acids (e.g. serine, proline, glutamine) can lead to microcephaly, developmental delay, seizures, spasticity and polyneuropathy. In adults, spastic paraplegia and polyneuropathies are to the fore.
Arginine and glutamine deficiency impair immune function and delay wound healing, particularly in the critically ill.
Muscle weakness and protein breakdown: chronic amino acid deficiency leads to muscle atrophy, hypoalbuminaemia and a higher risk of infection.
ORY OneCare Prevent – prevention starts with diagnostics
Become a partner now & offer diagnostics to your patients
*Evidence-based research
Stabler SP. The New England Journal of Medicine. 2013;368(2):149-60. doi:10.1056/NEJMcp1113996.
Green R, Miller JW. Vitamins and Hormones. 2022;119:405-439. doi:10.1016/bs.vh.2022.02.003.
3.Cobalamin Deficiency: Clinical Picture and Radiological Findings.
Briani C, Dalla Torre C, Citton V, et al.
Nutrients. 2013;5(11):4521-39. doi:10.3390/nu5114521.
4.Alterations in Sulfur Amino Acids as Biomarkers of Disease.
Stabler SP. The Journal of Nutrition. 2020;150(Suppl 1):2532S-2537S. doi:10.1093/jn/nxaa118.
5.Cobalamin, Folic Acid, and Homocysteine.
Varela-Moreiras G, Murphy MM, Scott JM.
Nutrition Reviews. 2009;67 Suppl 1:S69-72. doi:10.1111/j.1753-4887.2009.00163.x.
Leading Journal
6.ACG Clinical Guideline: Malnutrition and Nutritional Recommendations in Liver Disease.
Singal AK, Wong RJ, Dasarathy S, et al.
The American Journal of Gastroenterology. 2025;120(5):950-972. doi:10.14309/ajg.0000000000003379.
Practice Guideline New Research
Gwathmey KG, Grogan J. Muscle & Nerve. 2020;62(1):13-29. doi:10.1002/mus.26783.
8.Vitamin Deficiencies in Children: Lessons From Clinical and Neuroimaging Findings.
Dupuy G, Roux CJ, Barrois R, et al.
European Journal of Paediatric Neurology : EJPN : Official Journal of the European Paediatric Neurology Society. 2024;50:6-15. doi:10.1016/j.ejpn.2024.02.013.
9.Amino Acid Synthesis Deficiencies.
de Koning TJ. Journal of Inherited Metabolic Disease. 2017;40(4):609-620. doi:10.1007/s10545-017-0063-1.
10.Acquired Amino Acid Deficiencies: A Focus on Arginine and Glutamine.
Morris CR, Hamilton-Reeves J, Martindale RG, Sarav M, Ochoa Gautier JB.
Nutrition in Clinical Practice : Official Publication of the American Society for Parenteral and Enteral Nutrition. 2017;32(1_suppl):30S-47S. doi:10.1177/0884533617691250.
11.Amino Acid Homeostasis and Signalling in Mammalian Cells and Organisms.
Bröer S, Bröer A. The Biochemical Journal. 2017;474(12):1935-1963. doi:10.1042/BCJ20160822.